<p>Kathryn Miller-Jensen of Yale University in the U.S. will test the hypothesis that latently infected HIV cells produce different protein phosphorylation signatures than uninfected cells in response to drug treatments. Identifying these latent HIV cells will enable the design of new therapies that selectively target and purge these latent reservoirs.</p>


Kathryn Miller-Jensen of Yale University in the U.S. will test the hypothesis that latently infected HIV cells produce different protein phosphorylation signatures than uninfected cells in response to drug treatments. Identifying these latent HIV cells will enable the design of new therapies that selectively target and purge these latent reservoirs.

Phosphorylation Signatures of Latently Infected T Cells

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